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Large Scale Targeting

BAC Targeting

Since 1998, ingenious targeting laboratory has completed over 2,500 gene targeting projects, including those using BAC recombineering, to preserve extensive regulatory elements and large genomic fragments exceeding 100 kilobases.

BAC targeting converts large genomic fragments into functional targeting vectors while maintaining complete regulatory architecture spanning 5 prime promoters, enhancer elements, coding sequences, and 3 prime regulatory regions.

2,500+
Projects Completed
100+
Kilobases Capacity
26+
Years Experience
800+
Publications

BAC Recombineering Fundamentals

BAC recombineering utilizes homologous recombination between short homologous sequences (typically 50 base pairs minimum) in E. coli strains expressing bacteriophage lambda recombination genes and Cre recombinase.

BAC Source Selection and Characterization

Research teams typically identify appropriate BAC clones from existing genomic libraries covering mouse, human, rat, or other organism genomes. Clone selection requires careful attention to insert structure to ensure BAC spans the complete genomic region of interest including all relevant regulatory elements.

Recombineering Cassette Insertion

Standard BAC recombineering procedures insert selection cassettes and homologous recombination arm sequences into BAC clones using FRT flanked cassettes for subsequent removal.

Large Genomic Fragment Preservation

BAC targeting excels for applications requiring preservation of complete regulatory elements controlling endogenous gene expression.

Copy Number and Integration Site Considerations

BAC inserts at safe harbor loci (H11, ROSA26, HPRT) typically integrate as single copy events when ES cell characterization confirms appropriate clones.

Internal Rearrangement Risk Assessment

Large genomic fragments carry inherent risk of internal rearrangement during bacterial propagation, recombineering procedures, or ES cell targeting. Careful strain selection using recombineering proficient strains minimizes risk.

ES Cell Targeting of Large Recombineered BACs

Targeting vector construction using recombineered BACs creates very large targeting constructs (often 200+ kilobases) requiring special electroporation and ES cell selection protocols.

Pre Germline Characterization Importance

Pre germline ES cell analysis becomes particularly critical for large insert targeting to detect internal rearrangement events originating during vector construction or ES cell targeting.

Junction Characterization

PCR and sequencing across insertion junctions between endogenous genome and transgene sequences confirms correct targeting at intended locus.

Timeline and Project Planning for BAC Targeting

BAC targeting projects involve complex allele design and verification. Contact us for current timeline estimates based on your specific project requirements.

1

Phase 1: BAC Selection and Characterization

Genomic library searches identify candidate BAC clones. Physical characterization via restriction mapping and gel electrophoresis confirms insert structure and boundaries.

2

Phase 2: Recombineering Vector Construction

Targeting vector sequences and functional cassettes insert into BAC clones via sequential recombineering rounds. Each round requires bacterial growth, selection, and characterization.

3

Phase 3: ES Cell Targeting and Selection

Targeting vectors transfect into ES cells, undergo selection, and yield correctly targeted clones at frequencies 10 to 100 fold lower than standard vectors.

4

Phase 4: Chimera Generation

Correctly targeted ES cell clones contribute to chimeric mice via blastocyst injection. Germline transmission verification through breeding establishes stable transgenic lines.

Applications Ideally Suited to BAC Targeting

Disease Modeling with Large Human Genomic Inserts

Researchers can insert disease associated human loci (100+ kilobases including regulatory elements) into mouse genomes while maintaining human genetic context relevant to disease mechanisms.

Regulatory Element Studies

Studies examining how multiple enhancers and silencers coordinate gene expression require BAC insert capacity to preserve these relationships in transgenic contexts.

Complex Transgenic Studies

Combining multiple functional elements (promoters, genes, reporters, regulatory cassettes) in defined spatial arrangements benefits from BAC recombineering flexibility.

Publications Utilizing BAC Targeting

Jiang Y, Sachdeva K, Goulbourne CN, Berg MJ, Peddy J, Stavrides PH, Pensalfini A, Pawlik M, Malampati S, Whyte L, Basavarajappa BS, Shivakumar S, Bleiwas C, Smiley JF, Mathews PM, Nixon RA. 2025. Increased neuronal expression of the early endosomal adaptor APPL1 leads to endosomal and synaptic dysfunction with cholinergic neurodegeneration J Neurosci 29(45): e2331242025

Serrano J, Boyd J, Brown IS, Mason C, Smith KR, Karolyi K, Maurya SK, Meshram NN, Serna V, Link GM, Gardell SJ, Kyriazis GA. 2024. The TAS1R2 G-protein-coupled receptor is an ambient glucose sensor in skeletal muscle that regulates NAD homeostasis and mitochondrial capacity Nat Commun 15(1): 4915

What Researchers Say

The Hephaestin flox model ingenious has made for us has been great. It has helped generate eight research publications.

Joshua Dunaief, PhD, MD

University of Pennsylvania

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Start Your BAC Large Scale Targeting Project

BAC recombineering enables preservation of complex regulatory elements and large genomic features critical for physiologically relevant transgenic models. Our experienced team can guide BAC selection, manage recombineering construction, oversee ES cell targeting, and deliver study ready mice efficiently.

Frequently Asked Questions

BAC inserts typically span 100 to 180 kilobases with practical capacity for full genomic features. Inserts exceeding 200 kilobases face declining insertion efficiency and increasing structural instability. Custom vector engineering may accommodate larger inserts for specific applications, though standard BAC protocols optimize for 100 to 150 kilobase inserts.

BAC DNA inherent instability in certain E. coli strains introduces deletions and rearrangements unpredictable in extent and location. Pre-targeting and post-targeting characterization through gel electrophoresis and restriction mapping detects gross alterations, though some internal changes may escape detection. Careful strain selection and handling minimize but cannot eliminate rearrangement risk.

Yes, researchers frequently construct disease model BAC inserts containing specific point mutations, deletions, or other variants identified in human disease. BAC recombineering permits introduction of precise sequence changes within large genomic context, enabling investigation of disease mechanisms within realistic regulatory environment.

PCR or qPCR confirms single copy integration. Junction PCR across integration boundaries provides sequence evidence of correct targeting.

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