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Cell Type Specific

Tissue Specific Knockout

Since 1998, ingenious targeting laboratory has completed over 2,500 mouse models, including conditional knockouts that enable tissue specific gene deletion. Our tissue specific knockout models have supported research published in over 800 peer reviewed publications, including articles in Science, Nature, and Cell.

Tissue specific knockout restricts gene deletion to defined cell types or organs while preserving gene function elsewhere in the organism. By crossing a floxed (conditional) allele to a tissue specific Cre driver line, gene deletion occurs only in cells where Cre recombinase is expressed.

2,500+
Projects Completed
800+
Publications
26+
Years Experience
100%
Success Rate

How Tissue Specific Knockout Works

This approach enables study of genes that cause embryonic lethality when deleted globally, isolation of cell autonomous gene functions, and dissection of tissue specific phenotypes without confounding systemic effects. A single floxed allele can be crossed to dozens of different Cre driver lines, enabling tissue specific deletion across virtually every organ system from one initial mouse line.

The Two Component System

Floxed (Conditional) Allele

The target gene contains LoxP sites flanking critical exons. The floxed allele functions normally in the absence of Cre recombinase.

Cre Driver Line

A transgenic or knockin line expressing Cre recombinase under a tissue specific promoter. Cre is expressed only in defined cell types.

Deletion Pattern Determination

The tissue specificity of gene deletion is determined entirely by the Cre driver:

Promoter activity:Which cells express the Cre transgene
Developmental timing:When Cre expression begins
Expression level:Efficiency of recombination
Mosaicism:Whether all target cells undergo deletion

Cre Driver Lines by Organ System

Nervous System

Cre DriverTarget PopulationTimingApplications
Nestin CreNeural progenitorsE10.5Pan neural deletion
Synapsin CreNeuronsPostnatalMature neuron function
CamKII CreForebrain excitatory neuronsPostnatalLearning, memory, behavior
DAT CreDopaminergic neuronsE15Parkinson models
GFAP CreAstrocytesE14.5Glial function
Neuroscience Mouse Models

Immune System

Cre DriverTarget PopulationTimingApplications
CD4 CreT cells (CD4+ and CD8+)DP thymocyteT cell development, function
LysM CreMacrophages, granulocytesMyeloid progenitorInnate immunity
CD19 CreB cellsPro B cellB cell development, function
CD11c CreDendritic cellsDC progenitorAntigen presentation
Foxp3 CreRegulatory T cellsTreg commitmentImmune tolerance
Immunology Mouse Models

Metabolic Tissues

Cre DriverTarget PopulationTimingApplications
Albumin CreHepatocytesE14.5Liver metabolism
Adiponectin CreAdipocytesAdipogenesisAdipose biology
Insulin CreBeta cellsE11.5Insulin secretion
MCK CreSkeletal musclePostnatalMuscle metabolism
Villin CreIntestinal epitheliumE12.5Gut function

Cardiovascular System

Cre DriverTarget PopulationTimingApplications
Myh6 (αMHC) CreCardiomyocytesE9Cardiac function
Tie2 CreEndothelial cellsE7.5Vascular biology
SM22α CreSmooth muscleE9.5Vascular smooth muscle
Cardiovascular Mouse Models

Choosing the Right Cre Driver

Key Selection Criteria

  • Specificity:Does Cre expression match your target population?
  • Efficiency:What percentage of target cells undergo recombination?
  • Timing:When does deletion occur relative to your experimental question?
  • Ectopic expression:Does the Cre driver have off-target expression?
  • Cre toxicity:High Cre levels can cause cellular effects independent of target gene deletion

Common Pitfalls to Avoid

  • Germline recombination:Some Cre drivers cause deletion in germline, converting conditional to global knockout
  • Leaky expression:Low level Cre in non-target tissues
  • Mosaic deletion:Incomplete recombination in target population
  • Developmental vs adult expression:Embryonic Cre deletes during development; may not reflect adult gene function

Applications

Essential Genes

Tissue specific knockout enables study of genes that cause embryonic lethality when deleted globally. By restricting deletion to non-essential tissues or adult stages, researchers can study gene function without developmental lethality.

Cell Autonomous Function

Tissue specific deletion distinguishes cell autonomous from non cell autonomous gene function. Does the phenotype arise from gene loss in the target tissue, or from secondary effects elsewhere?

Target Validation

Tissue specific knockout reveals where target inhibition would have therapeutic effects and where it might cause toxicity, supporting drug development decisions.

Selected Publications

Salzbank J, Lacaille H, Gaby J, O'Reilly JJ, Kissner M, Vacher CM, Penn AA 2025. Microglia alter sex-specific cerebellar myelination following placental hormone loss.

Nat Commun. 16(1): 9846

Wang L, Noyer L, Jishage M, Wang YH, Tao AY, McDermott M, Gando I, Sidhu I, Hu K, Zhong L, Sun K, Drmic D, Kaufmann U, Feske S 2025. CLNS1A regulates genome stability and cell cycle progression to control CD4 T cell function and autoimmunity.

Sci Immunol 108(10): eadq8860

Milanick W, Li J, Thomas CI, Al-Yaari M, Guerrero-Given D, Kamasawa N, Young SM Jr 2025. Presynaptic α2δs specify synaptic gain, not synaptogenesis, in the mammalian brain.

Neuron 12(113): p1886-1897.E9

View All Publications

What Researchers Say

ingenious Targeting Laboratory is highly recommended for generating custom animal models. Past 2 years, we have made 2 conditional knockout mouse lines. All processes of each project were scientifically and professionally handled. Their scientific consulting to initiate the project was superb compared to other companies, and transparency of the project progress reported by project managers was excellent. Their excellency and dedication to meet our needs in a timely manner are invaluable to continuation of our research progress.

Hyekyung Plumley, PhD

Warren Center for Neuroscience Drug Discovery

Start Your Tissue Specific Knockout Project

Our scientific consultants are ready to discuss your research requirements and the optimal floxed allele design for your experimental goals. Initial consultation is provided at no charge.

✦ New for 2026

Breeding Scheme Architect

Plan complex multi-allele breeding strategies, calculate expected genotype ratios, and estimate time to experimental cohorts—all before starting your project.

Visualize multi-generation breeding paths
Calculate Mendelian ratios automatically
Estimate timeline to study-ready cohorts

Free Research Tool

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Allele 1Gene-flox (conditional)
Allele 2Cre-driver (tissue-specific)
TargetHomozygous knockout

→ 3 generations to target genotype

Frequently Asked Questions

Tissue-specific knockouts combine floxed alleles (LoxP sites flanking critical exons) with Cre driver lines that express Cre recombinase in specific cell types or tissues. When the floxed mouse is crossed to a tissue-specific Cre driver, gene deletion occurs only in Cre-expressing tissues while gene function is preserved elsewhere. This enables study of tissue-autonomous gene function.

Common Cre drivers include Albumin-Cre (hepatocytes), Nestin-Cre (neural progenitors), LysM-Cre (myeloid cells), CD4-Cre (T cells), Adiponectin-Cre (adipocytes), Myh6-Cre (cardiomyocytes), and many others. Selection depends on your target tissue and research question. Reporter crosses verify expected tissue distribution before experimental use.

Yes. This is a major advantage of conditional knockout systems. A single floxed allele can be crossed to any Cre driver line to achieve tissue-specific knockout in different organs. One floxed mouse project can support studies across multiple tissues (liver, brain, immune cells, etc) simply by crossing to different Cre drivers.

Required controls include: floxed mice without Cre (flox/flox; +/+) as baseline controls, Cre-only controls (+/+; Cre/+) to assess Cre effects, and tissue-specific knockout experimental animals (flox/flox; Cre/+). Comparing these groups ensures that phenotypes result from gene deletion in the target tissue rather than Cre expression or background effects.

Genomic PCR using primers flanking the LoxP sites reveals deletion: wildtype and floxed alleles produce larger bands; recombined (deleted) allele produces a smaller band. Reporter crosses (Rosa26-tdTomato) visualize recombination pattern and confirm deletion in expected tissues. Functional assays (Western blot, immunohistochemistry) confirm loss of protein in target tissue.

Lab Signals

Tissue Specific Knockout Insights

Explore Cre driver selection strategies, tissue-specific targeting approaches, and conditional knockout best practices. Expert analysis delivered biweekly.

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