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Brain & Behavior Research

Neuroscience Mouse Models

Since 1998, ingenious targeting laboratory has supported neuroscience researchers with custom mouse models contributing to peer reviewed publications in Nature Neuroscience, Neuron, Cell, Journal of Neuroscience, and leading neuroscience journals worldwide.

Our neuroscience mouse models have advanced understanding of neural development, synaptic function, behavior, and neurological disease.

2,500+
Projects Completed
800+
Publications
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Years Experience
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Neuroscience mouse models enable researchers to investigate the molecular and cellular mechanisms underlying brain function and dysfunction. From cell type specific knockouts that dissect neural circuit function to disease mutation knockins that model neurological conditions, the right model design is critical for understanding the nervous system and developing treatments for neurological disorders.

Conditional approaches are particularly important in neuroscience, where the same gene often has distinct functions in different neuronal populations, glial cells, and developmental stages. ingenious targeting laboratory designs and generates custom neuroscience models tailored to your specific research questions.

Cell Type Specific Approaches

Neuronal Cre Drivers

Conditional knockouts enable gene deletion in specific neuronal populations:

Cre DriverTarget PopulationTimingApplications
Nestin CreNeural progenitorsE10.5Pan neural knockout
Synapsin CreMature neuronsPostnatalPost mitotic neuron function
CamKIIα CreForebrain excitatory neuronsP14+Learning, memory, cognition
Emx1 CreCortical excitatory neuronsE10.5Cortical development, function
DAT CreDopaminergic neuronsE15Reward, movement, Parkinson
Chat CreCholinergic neuronsE12.5Motor function, cognition
Pvalb CreParvalbumin interneuronsPostnatalInhibitory circuits, oscillations
Sst CreSomatostatin interneuronsPostnatalCortical inhibition
Vgat CreGABAergic neuronsE12.5All inhibitory neurons
Vglut2 CreGlutamatergic neuronsE11.5Excitatory transmission

Glial Cre Drivers

Cre DriverTarget PopulationTimingApplications
GFAP CreAstrocytesE14.5Glial function, gliosis
Aldh1l1 CreAstrocytesPostnatalMore specific astrocyte targeting
Olig2 CreOligodendrocyte lineageE12.5Myelination
PLP CreMature oligodendrocytesPostnatalMyelin maintenance
CX3CR1 CreMicrogliaEmbryonicNeuroinflammation

Inducible Neural Cre Lines

Tamoxifen inducible Cre (CreERT2) enables temporal control over gene deletion in the nervous system:

CamKIIα CreERT2:Inducible deletion in forebrain excitatory neurons
Nestin CreERT2:Inducible deletion in neural progenitors
GFAP CreERT2:Inducible deletion in astrocytes
PLP CreERT2:Inducible deletion in oligodendrocytes

Inducible systems enable adult onset gene deletion, bypassing developmental requirements and modeling acute loss of function.

Neurodegenerative Disease Models

Alzheimer Disease

Models addressing amyloid and tau pathology:

  • APP knockin models expressing familial AD mutations
  • Tau knockin and humanization models
  • Presenilin knockout and knockin models
  • Microglial gene modifications for neuroinflammation studies
Learn more

Parkinson Disease

Models for dopaminergic neuron biology and alpha synuclein pathology:

  • Alpha synuclein knockin and overexpression models
  • LRRK2 mutation knockins
  • PINK1, Parkin, DJ1 knockouts for mitochondrial dysfunction
  • DAT Cre mediated conditional knockouts in dopaminergic neurons
Learn more

ALS and Motor Neuron Disease

Models for motor neuron degeneration:

  • SOD1 mutation knockins (G93A, G85R)
  • TDP43 and FUS mutation models
  • C9orf72 repeat expansion models
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Huntington Disease

Models for polyglutamine expansion pathology:

  • HTT knockin models with expanded CAG repeats
  • Conditional HTT knockouts
  • Striatal specific models using D1R or D2R Cre drivers
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Research Applications

Neural Circuit Dissection

Cell type specific knockouts enable circuit level analysis:

  • Identify which neurons require specific genes for circuit function
  • Distinguish cell autonomous from circuit level effects
  • Map gene function to specific neural populations
  • Combine with optogenetics and chemogenetics

Synaptic Function

Models for studying synaptic transmission and plasticity:

  • Receptor knockout and knockin models
  • Synaptic vesicle and release machinery modifications
  • Postsynaptic density protein studies
  • Synaptic plasticity gene modifications

Behavioral Studies

Models supporting behavioral neuroscience:

  • Learning and memory (hippocampal, cortical)
  • Anxiety and depression (limbic circuits)
  • Motor function (basal ganglia, cerebellum)
  • Social behavior (prefrontal, amygdala)
  • Reward and addiction (VTA, nucleus accumbens)

Developmental Neuroscience

Models for neural development studies:

  • Neural progenitor gene function
  • Neuronal migration and differentiation
  • Axon guidance and synaptogenesis
  • Critical period plasticity

Model Types for Neuroscience

Conditional Knockout

Essential for neuroscience due to:

  • Many neural genes cause embryonic lethality when deleted globally
  • Cell type specificity reveals circuit level gene function
  • Temporal control distinguishes developmental from adult roles
  • Brain region specificity possible with appropriate Cre drivers
Learn more

Point Mutation Knockin

Model disease associated variants at physiological levels:

  • Familial disease mutations (AD, PD, HD)
  • GWAS identified risk variants
  • Phosphorylation site mutations for signaling studies
Learn more

Reporter Knockin

Visualize gene expression and cell populations:

  • Cell type markers for identification and sorting
  • Activity reporters for functional imaging
  • Lineage tracing of neural progenitors
Learn more

Technical Considerations

Strain Background

Strain background affects neural and behavioral phenotypes:

C57BL/6:Most common, good learning performance, some anxiety
129 strains:Variable behavioral phenotypes, less commonly used
BALB/c:Distinct behavioral profile, higher anxiety
FVB:Good for transgenics but retinal degeneration (rd1)

C57BL/6 is generally preferred for neuroscience to enable comparison with published literature and behavioral databases.

Behavioral Phenotyping Considerations

  • Age matching for behavioral cohorts
  • Sex as biological variable
  • Circadian timing of testing
  • Environmental standardization
  • Appropriate statistical power

Selected Publications

Neuroscience models generated by ingenious targeting laboratory:

Vacher CM et al. (2021).

Placental endocrine function shapes cerebellar development and social behavior.

Nature Neuroscience 24(10): 1392-1401

Chakrabarti S et al. (2024).

Touch sensation requires the mechanically gated ion channel ELKIN1.

Science 383(6686): 992-998

Clausen BE et al. (1999).

Conditional gene targeting in macrophages and granulocytes using LysMcre mice.

Transgenic Research 8(4): 265-277

✦ New for 2026

Breeding Scheme Architect

Plan complex multi-allele breeding strategies, calculate expected genotype ratios, and estimate time to experimental cohorts—all before starting your project.

Visualize multi-generation breeding paths
Calculate Mendelian ratios automatically
Estimate timeline to study-ready cohorts

Free Research Tool

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Allele 1Gene-flox (conditional)
Allele 2Cre-driver (tissue-specific)
TargetHomozygous knockout

→ 3 generations to target genotype

What Researchers Say

ingenious Targeting Laboratory is highly recommended for generating custom animal models. Past 2 years, we have made 2 conditional knockout mouse lines. All processes of each project were scientifically and professionally handled. Their scientific consulting to initiate the project was superb compared to other companies, and transparency of the project progress reported by project managers was excellent. Their excellency and dedication to meet our needs in a timely manner are invaluable to continuation of our research progress.

Hyekyung Plumley, PhD

Warren Center for Neuroscience Drug Discovery

Start Your Neuroscience Model Project

Our scientific consultants are ready to discuss your neuroscience research requirements and recommend the optimal model design and Cre driver selection for your program. Initial consultation is provided at no charge.

Frequently Asked Questions

Many neural genes are essential for embryonic development and cause lethality when deleted globally. Conditional knockouts allow gene deletion in specific cell types (neurons, astrocytes, microglia) or brain regions, enabling study of gene function in adult animals without developmental defects.

Common neural Cre drivers include Nestin-Cre (neural progenitors), Synapsin-Cre (neurons), GFAP-Cre (astrocytes), and CX3CR1-Cre (microglia). Inducible CreERT2 systems enable temporal control. Cre driver selection depends on your target cell type and research question.

Yes. Point mutation knockins can introduce human disease mutations (e.g., APP, tau, α-synuclein mutations for AD, PD). Conditional systems enable tissue-specific expression of disease proteins. Reporter knockins enable visualization of disease protein aggregation and spread.

C57BL/6 is most commonly used for behavioral studies due to good learning performance, extensive characterization, and compatibility with published behavioral databases. BALB/c and 129 strains have distinct behavioral profiles that may be appropriate for specific research questions.

Standardize age, sex, circadian timing, and environmental conditions. Use littermate controls from heterozygous crosses. Ensure adequate statistical power. Consider that genetic background modifiers can affect behavioral outcomes, so maintaining defined backgrounds through backcrossing is important.

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